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Southern Research Institute to Present at the 2007 American,Association For Cancer Research Annual Meeting

BIRMINGHAM, Ala., April 03, 2007 /PRNewswire/ -- Southern Research Institute, a not-for-profit organization that conducts basic and applied research in the areas of preclinical drug discovery and drug development today announced that its scientists will be delivering one oral presentation and 10 poster presentations at the 2007 American Association for Cancer Research (AACR) Annual Meeting, April 14-18 in Los Angeles, Calif.

Anshu Mittal Roy of Southern Research will be presenting new data on the different factors that contribute to tumor growth. The discussion titled "VEGF Overexpression Increases Stromal Content in Both Primary Tumor and Metastatic Lesions" will be presented on Tuesday, April 17, from 11:25 a.m. - 11:40 a.m. in Room 404 A-B, Los Angeles Convention Center.

Scientists from Southern Research will also present preclinical data in posters at the Exhibit Hall:

    Sunday, April 15

     -- Jose T. Thaiparambil will be presenting poster (#644) "Sulindac

        Sulfide Selectively Inhibits cGMP PDE from Human HT-29 Colon Tumor

        Cells" from 8:00 a.m. -12:00 p.m.


     -- Patricia E. Noker will be presenting poster (#1545) "Toxicity and

        Pharmacokinetics of 1-Methyl-D-Tryptophan in Rats and Dogs" from 1:00

        p.m. - 5:00 p.m.


    Monday, April 16

     -- Rongbao Li will be presenting poster (#3165) "Structural Basis for

        PYK2 Adhesion Targeting" from 1:00 p.m. - 5:00 p.m.


     -- Adam B. Keeton will be presenting poster (#2282) "Reversal of MRP-1

        Mediated Multi-Drug Resistance by Sulinda Sulfide and Indomethacin"

        from 8:00 a.m. - 12:00 p.m.


    Tuesday, April 17

     -- Bo Xu will be presenting poster (#3895) "Regulation of ATM

        Phosphorylation in the Absence of DNA Damage" from 8:00 a.m. - 12:00

        p.m.


     -- Mickael J. Cariveau will be presenting poster (#3893) "Interfering

        with NBS1-ATM Interaction to Inhibit the DNA Damage Response and

     
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