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Samaritan's Alzheimer's Drug Caprospinol Suggests Brain May be Able,to Heal From Alzheimer's Disease


Animal Study Confirms Caprospinol's (SP-233) Ability to Clear Brain Plaque and Empower Rat Brain Cells to Recover Memory Function

In Vivo 'Proof of Concept' Study Proposes Direct Correlation Between Removing Beta Amyloid Plaque and Memory Recovery

LAS VEGAS, July 10, 2007 (PRIME NEWSWIRE) -- Samaritan Pharmaceuticals Inc. (AMEX:LIV), a developer of innovative drugs, is pleased to announce it has confirmed Caprospinol's (SP-233) efficacy in its ability to dramatically decrease beta amyloid plaque in the brain, accompanied by a complete recovery of memory back to baseline (p=0.002), as shown in a Morris water maze task. This study confirms previously reported in-vitro data and suggests Caprospinol may not only slow down Alzheimer's but may also reverse plaque-related brain injuries associated with the mind-robbing disease of Alzheimer's.

The hallmark of Alzheimer's disease is plaque formation in areas of the brain that control memory and thinking skills. Plaque can cause swelling and bumps on nerve cells, making it hard or even impossible for the cells to communicate. While the cause of Alzheimer's disease still remains unknown, a growing number of researchers are indicating that plaque formation and accumulation may play a role.

The study was conducted by Samaritan's collaborating researchers, including Dr. Laurent Lecanu and Dr. Vassilios Papadopoulos.

Abstract:

www.samaritanpharma.com/caprospinol_abstract.pdf

Dr. Janet Greeson, CEO of Samaritan Pharmaceuticals stated, "Memory recovery in rats is a lot simpler than curing Alzheimer's disease in people but these study results are extremely encouraging. The rats tested with our proprietary Alzheimer's rat model took on features similar to the crippling dementia seen in a human's Alzheimer's brain, and then, by treating the
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