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Studie von Grifols zeigt, dass eine höhere Dosis von PROLASTIN-C den Alpha1-PI-Spiegel in Patienten mit AAT-Mangel in den normalen Bereich anhebt
Date:5/22/2013

SDAQ mit ADRs (NASDAQ: GRFS) gehandelt. Weitere Informationen finden Sie unter www.grifols.com.

Über Alpha1-Antitrypsinmangel
Alpha1-Antitrypsinmangel, auch als AAT-Mangel oder Alpha-1 bekannt, ist eine vererbte Krankheit, die zu einer nennenswerten Reduktion eines natürlich vorkommenden Proteins, des Alpha1-Proteinasehemmers, führt. Alpha1 ist zwar selten, aber die häufigste Ursache von genetischem Emphysem in Erwachsenen und die häufigste Ursache von Lebererkrankungen in Kindern. Personen, die an Alpha1 leiden, entwickeln sehr oft schwere chronisch obstruktive Lungenerkrankungen (COPD), die zu Behinderungen und vorzeitigem Tod führen. Man schätzt, dass in Nordamerika und Europa insgesamt 200.000 Menschen von Alpha1 betroffen sind, und dass die Krankheit bei über 90 % der Betroffenen nicht erkannt wird.

Über PROLASTIN® und PROLASTIN®-C

PROLASTIN und PROLASTIN-C sind für die Behandlung von Alpha1-Antitrypsinmangel (AAT) in Patienten mit Emphysem indiziert. AAT-Mangel ist eine genetische Störung, bei der der niedrige Spiegel des Alpha1-Proteins bei Erwachsenen zu Emphysem und vorwiegend bei Kindern zu Leberkrankheiten führen kann. Das aktive Protein in PROLASTIN und PROLASTIN-C erhöht bzw. steigert den Proteinspiegel in Patienten mit in AAT-Mangel. PROLASTIN ist in einigen Ländern Europas und Südamerikas zugelassen, während PROLASTIN-C in den USA, in Kanada, Argentinien und Kolumbien zugelassen ist.

Wichtige Sicherheitshinweise zu PROLASTIN®-C

PROLASTIN-C, Alpha1-Proteinasehemmer (human), ist zur chronischen Augmentations- und Erhaltungstherapie bei Erwachsenen mit Emphysemen aufgrund eines Alpha-1-Proteinasehemmer-Mangels (Alpha-1-Antitrypsinmangel) indiziert.

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