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New ADHD/Retina Research Supports a Cerebellar Origin, states Dr. Harold Levinson
Date:6/21/2013

NEW YORK, June 21, 2013 /PRNewswire-iReach/ -- A highly original ADHD study that was just presented at the American Psychiatric Association's 2013 Annual Meeting offers new and intriguing diagnostic possibilities about ADHD/retina research. In addition, this study's finding supports the cerebellar signal-scrambling theory of dyslexia and related ADHD. This theory was initially proposed by Dr. Harold Levinson and was supported by Nobel Laureate cerebellar neurophysiologist Sir John Eccles.

(Photo: http://photos.prnewswire.com/prnh/20130621/CG36262)

On May 18, 2013, Emanuel Bubl, MD presented evidence indicating an alteration in visual signal transmission, perception and elevated noise within the retina's ganglion cells of ADHD patients versus normal controls. A simple EKG-like recording of retinal neuronal activity called a pattern electroretinogram (PERG) was used and appears to have biological diagnostic potential in ADHD.

Sir John Eccles was awarded the Nobel Prize for discovering that the cerebellum actively inhibits, filters and thus modulates all neuronal signals at their receptor sites—for example, the retina's ganglion neurons. A cerebellar dysfunction would then clearly explain the retinal findings reported by Dr. Bubl at the APA.

Dr. Levinson merely expanded Eccles' cerebellar concepts, reasoning that the cerebellum modulates all brain signals. These patient-derived concepts, now consistent with advanced cerebellar studies, have been detailed in research papers as well as in his book "Total Conce
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SOURCE Levinson Medical Center for Learning Disabilities
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