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Long-Term Data on Cimzia® (certolizumab pegol) for Moderate to Severe Crohn's Disease to be Highlighted at ACG Meeting
Date:10/15/2010

ATLANTA, Oct. 15 /PRNewswire/ -- Studies and analyses of the Crohn's disease (CD) treatment Cimzia® (certolizumab pegol) will be exhibited at the 2010 Annual Scientific Meeting of the American College of Gastroenterology, taking place in San Antonio from October 15 – 20.  

"Cimzia data at this year's ACG meeting continue to demonstrate real-life significance to those with moderate to severe Crohn's disease who have tried other therapies with no response or have never been treated with an anti-TNF," said Cem Kayhan, MD, Medical Director at UCB.  "These data indicate Cimzia may be beneficial to these patient populations in many aspects, including rapid relief of symptoms, long-term remission maintenance and quality of life improvements."

Individuals with moderate to severe Crohn's disease can experience painful symptoms that could interfere with daily life such as joint pain, abdominal cramps and high fevers.  Cimzia is indicated for reducing the signs and symptoms of Crohn's disease and maintaining clinical response in adult patients with moderately to severely active disease who have had an inadequate response to conventional therapy.

Posters highlighting results from multiple trials demonstrate the utility of Cimzia in the treatment of Crohn's disease patients:

  • Endoscopic Improvement in Patients with Active Crohn's Disease Treated with Certolizumab Pegol of Blinded Central Reading of Recorded Endoscopies from the MUSIC Study
  • Central reading of recorded endoscopies confirmed significant improvement in mucosal healing based on CD Endoscopic Index of Severity (CDEIS) scores at Week 10 in patients with active CD who were treated with certolizumab pegol (CZP).  
  • Poster 285, October 17, 3:30 – 7:00 pm, Exhibit Hall CD
  • Health-Related Quality of Life Improvements in Patients with Active Crohn's Disease Following Treatment with Certolizumab Pegol in the MUSIC Study
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  • SOURCE UCB
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