Genetic Profile Identified for Alzheimer's Patients Showing Heightened Response to Accera's Breakthrough Prod... (rprising because the majority of partici...)

Genetic Profile Identified for Alzheimer's Patients Showing Heightened Response to Accera's Breakthrough Product Axona®

Date:10/17/2011

rprising because the majority of participants in the study were already taking one or more approved Alzheimer's disease medications. "More than two dozen clinical trials have demonstrated that certain therapies offer clear genotype dependent clinical improvements," commented Dr. Judes Poirier of McGill University. Dr. Poirier is internationally renowned for his works on the role of apolipoprotein E in the normal and injured brain, and in the genetics of Alzheimer's disease. According to Dr. Poirier, "this study takes this notion to the next level, linking multiple genetic risk factors to significant therapeutic response in a state-of-the-art, randomized, double-blind, placebo controlled study in mild-to-moderate Alzheimer's disease."

Alzheimer's disease is characterized by decreases in the brain's ability to metabolize glucose, a well recognized condition known as hypometabolism. Since glucose is the predominant fuel used by the brain, disruptions in glucose metabolism are associated with cognitive impairment. Axona, a medical food product taken once daily, overcomes this deficiency by providing the brain with ketone bodies, an alternative fuel source that results in improved cognition.

Dr. Richard Isaacson, Associate Professor of Clinical Neurology at the University of Miami Miller School of Medicine and author of the book "Alzheimer's Treatment / Alzheimer's Prevention: A Patient and Family Guide" also finds these results encouraging. Dr. Isaacson explains that "clinicians have been struggling to understand why certain patients with Alzheimer's disease may preferentially respond to one therapy, while other patients will not respond. This exploratory study is important in that it takes the first steps toward understanding how specific genes may influence the response to ketosis therapy, and improve our ability to address glucose hypometabolism in the brain."

The title of the article is "Pharmacog
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SOURCE Accera, Inc.
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