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Epidemics of Both Type 1 Diabetes (Insulin Dependent) and Type 2 Diabetes (Obesity Related) Are Linked to Immunization
Date:12/10/2008

BALTIMORE, Dec. 10 /PRNewswire/ -- Data by Dr. J. Bart Classen published this week in Diabetes & Metabolic Syndrome: Clinical Research & Reviews provides further evidence that epidemics of type 2 diabetes/obesity/metabolic syndrome, like type 1 diabetes, are linked to immunization. Classen previously published proof vaccines are causing an epidemic of type 1 diabetes in children.

The new data as well as Classen's recently published data demonstrate that the epidemics of type 1 diabetes and type 2 diabetes/obesity/metabolic syndrome in children are linked. Exposure to vaccines causes some individuals to develop an autoimmune disease such as type 1 diabetes. In other individuals vaccine induced inflammation is countered by release of cortisol and other factors to suppress the inflammation. The release in cortisol and other factors leads to a "cushingoid" like state and the development of type 2 diabetes/obesity/metabolic syndrome.

Classen's current paper shows that those races which have high cortisol activity, especially after immunization, have a low risk for developing type 1 diabetes but a high risk for developing type 2 diabetes. Classen has previously demonstrated vaccine induced type 1 diabetes has a strong genetic/familial risk and those who have a sibling with type 1 diabetes have a much greater risk of developing vaccine induced type 1 diabetes.

In a previous publication in The Open Endocrinology Journal, Dr. Bart Classen showed a 50% reduction of type 2 diabetes occurred in Japanese children following the discontinuation of a single vaccine, a vaccine to prevent tuberculosis. This decline occurred at a time when there is a global epidemic of type 2 diabetes and metabolic syndrome.

"The picture is becoming clear. Not only are vaccines causing an epidemic of autoimmunity including type 1 diabetes but they are causing an epidemic of metabolic syndrome as
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SOURCE Classen Immunotherapies, Inc.
Copyright©2008 PR Newswire.
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