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Dermagen Reports Successful Phase I/IIa Clinical Trial for Atopic Dermatitis
Date:12/2/2009

r exacerbating factor in AD. Superantigens produced by the bacteria stimulate keratinocytes and T-lymphocytes, and trigger the inflammatory process. The inflammation leads to an impaired skin barrier function. Furthermore, endogenous antimicrobial peptides are low in patients with AD, leading to a vicious cycle of S. aureus involvement in the disease. Superantigens also act as potent inhibitors of steroid action, possibly leading to steroid resistance.

The use of topically administered AMPs for control and normalization of the bacterial and fungal flora is therefore a logical and attractive method for interfering with the pathogenesis of AD. It is well established that treatment of AD with steroids in combination with antibiotics is more effective than use of steroids alone. In the light of current therapies, endogenous AMPs will add an important new and significant therapeutic modality to current treatments with a very low probability of inducing bacterial resistance.

    For further information, please contact:

    Jan Alenfall, Managing Director, DermaGen AB
    Phone: +46(0)46-192-197 / Mobile: +46(0)709-315-115
    E-mail: jan.alenfall@dermagen.se

    Jorgen Thorball, Chairman, Pergamum AB
    Phone: + 45(0)307-786-27
    E-mail: jorgen.thorball@pergamum.com

    Conny Bogentoft, CEO, Karolinska Development AB
    Phone: +46(0)70-668-61-43
    E-mail: conny.bogentoft@karolinskadevelopment.com

SOURCE DermaGen AB


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SOURCE DermaGen AB
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