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Targeting SOCS-3 can Control Obesity

n that regulates appetite and body weight. Meanwhile, other portions of the weight regulation system remained intact and in fact became more responsive, thereby suggesting that arcuate nucleus function is the point of breakdown during leptin deficiency.

Finally the research highlighted a key gene called SOCS-3 involved in leptin deficiency. By targeting the gene with therapeutics, scientists may be able to repair leptin deficiency, aiding in weight loss.

"Of course these findings are quite far from the development of a weight loss drug," explained Cowley. "This work further highlights the essential role of SOCS-3 in this critical regulation system."

"One of the surprising things was when we put the genetically identical mice on the high fat diet, some became obese, and some did not. One major difference between these groups of mice was the activity of SOCS-3," said Pablo Enriori Ph.D. of Cowley's lab, first author of the paper. The research was conducted in conjunction with Brown University in Rhode Island.

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