( ith the David Geffen School of Medicine...)Success of HIV may depend in its mutations ,Health

ith the David Geffen School of Medicine at the University of California, Los Angeles, told UPI. "This shows that the response to virus is lost later in infection. Now we need to know why," she said. "While there are no direct implications for diagnosis or treatment, an important motivation for this sort of study is the development of knowledge that could lead to successful HIV vaccines, both to prevent and to treat HIV infection," infectious disease specialist Stuart Ray, of the Johns Hopkins University School of Medicine in Baltimore, told UPI. When a person is first infected with HIV, his or her immune system responds by producing killer T-cells, called CD8- T, and CD4-T helper cells. The system also remembers the type of molecules on the surface of the attacking virus, so the next time the system encounters the virus, it produces many cells from memory to attack it again. But when the virus mutates, the immune system is no longer able to identify and kill the mutations.

The researchers found much of the deficiency also occurs because HIV destroys the helper cells. This stops the efficient production of new killer cells and it helps to weaken the killer cells that are produced. Because of this finding, Jamieson recommended further research "looking at the epitopes (surface proteins) on the virus over time in individuals."


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