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Study Finds Astaxanthin Is Potent Inhibitor of Oxidation in Cell Membrane Model

> In addition, by limiting the amount of oxidative damage to LDL, the “bad cholesterol,” an astaxanthin derivative could alter the progress of atherosclerosis, the underlying inflammatory process that clogs arteries and is responsible for most heart attacks and strokes when an atherosclerotic plaque ruptures.

Pashkow maintains that statins represented the “first big wave” of cardiovascular preventive therapy. They proved to lower the risk of a second heart attack or cardiovascular death by 45% over 5 years, though more than half of the people treated did not realize their benefit. Increasing the dose of the statin improves the outcome and extends the benefits to people who have risk factors alone such as high cholesterol, but dose limiting side effects emerge especially when the drugs are taken for a long time. Pashkow said that the recent problems encountered with Pfizer’s torcetrapib highlight the impact of a late stage pipeline non-approval combined with the looming patent expiry of the current market leading therapy.

Antiplatelet therapies, particularly aspirin and aspirin in combination with other antiplatelet agents represent the “second big wave” of preventive therapies following a heart attack. In combination, they reduce the risk of future heart attacks and strokes by about 30% over a one-year period.

However, an appreciable segment of the population are 'resistant' to antiplatelet therapy and remain relatively unprotected. More potent antiplatelet therapies, or increased doses of aspirin increase the risk of bleeding. “This wave of treatments,” said Pashkow “may be peaking as well.”

Cardax Pharmaceuticals is exploring the use of astaxanthin and novel astaxanthin derivatives, as well as other carotenoids, for use as medical therapeutics (i.e. antioxidant/anti-inflammatory) in disease states characterized by abnormal cellular oxidation and chronic inflammation (e.g. cardiovascular, liver, ocular, prostatic and
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