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Schizophrenia Complicates With Altered Receptor Activity

hesizes that schizophrenia may be, in part, caused by the convergence of multiple factors (both genetic and epigenetic) at the PSD, which, in turn, alters the interaction of the molecules in the cellular environment, resulting in the symptoms of schizophrenia. In a continued attempt to understand the differences between the brains of mentally healthy patients and those with schizophrenia, future studies by the research team at Penn will focus on identifying differences in interactive dynamics of proteins in the PSD.

The postmortem brain stimulation method, established in this study, breaks out of the boundaries of previous research using postmortem brain tissue. Postmortem studies have historically focused on snapshot analyses of the brain at the time of death. This new method allows investigation of functional responses of brain tissue to stimulation. ‘Our hope is that this study will shift our postmortem methodologies from limited comparative studies to a more experimental approach,’ explains Arnold. ‘This will allow us to tease apart the molecular complexities that contribute to mental illnesses such as schizophrenia.’

(Source: Erekalert)
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