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Protein Increase Decreases Risk of Heart Failure

vels in the heart after heart attack.

Certain cell receptors called Gq-coupled receptors, which include alpha-adrenergic and angiotensin receptors, play a role in cardiac hypertrophy and "have been implicated in being elevated in some models of heart disease," he says. "When these agents act on the heart, they stimulate pathways that lead to apoptosis and cardiac hypertrophy. It seems that they also lead to changes in S100A1 expression.

"It's clear that whenever you want to manipulate a heart molecule that doesn't have an enzymatic function, we have to use gene therapy," he notes. "We want to know as much about this as possible in case we can come up with a small molecule that can regulate its expression.

"Perhaps antagonists that act on Gq-coupled receptors, which are being used clinically for heart failure, may be part of the mechanism of action in heart failure, which is why they appear to give beneficial results. They prevent further S100A1 decreases through this mechanism."

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