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Osteoporosis – A New Treatmen

atments for osteoporosis, Crabtree said. Very little NFATc1 must be activated to build extra bone, Winslow noted, which means that it may be possible to up-regulate the calcineurin/NFATc pathway to promote bone formation without disturbing other organ systems that use this same pathway.

‘The results were dramatic, yet the molecular alteration is very, very minimal,’ Crabtree said. NFATc1 in the mice that developed extra bone mass was only 10 percent more active than it is in normal mice.

The researchers are now screening chemical libraries for small molecules that could increase NFATc just enough to promote bone formation in people with osteoporosis, without causing undesirable side effects. ‘If you could find a small molecule that would flip 10 percent of the existing NFATc into the active form,’ Crabtree said, ‘you could favor the formation of osteoblasts and make stronger bones.’

(Surce: Eurekalert)
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