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Leukemia Gets A New Drug

ormal activation of FIP1L1-PDGFRá. This new research is being published this week on the website of the scientific journal PNAS. Resistance to the remedy In the fight against CEL (and other forms of leukemia), scientists use proteins that inhibit the tyrosine kinases. Glivec is such an inhibitor and is effective against CEL because it specifically inhibits the activity of FIP1L1-PDGFRá. However, CEL patients must take Glivec every day for the rest of their lives − and recent research shows that, over time, alterations in the DNA can arise, causing resistance to Glivec. The longer Glivec is taken, the greater the chance resistance will develop. At that point, treatment with Glivec is no longer effective. On the path to a long-lasting effective treatment This problem prompted VIB researchers Els Lierman and Jan Cools to look for alternatives. They have found that Sorafenib, another inhibitor, works effectively in treating the resistant form of CEL. Sorafenib is already on the market in the US as a remedy for kidney tumors. This new research indicates that, to be able to treat certain forms of leukemia (like CEL) effectively over a long period of time, several inhibitors must be used, either together or successively. The scientists emphasize the importance of testing known inhibitors for their effectiveness against CEL and other forms of leukemia. This research has recently appeared in the scientific journal Blood. Contact: Sooike Stoops info@vib.be 329-244-6611 VIB, Flanders Interuniversity Institute of Biotechnology Source: Eurekaler
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