commended optimal level for HbA1c.
The individuals in the study reported that they slept an average of six hours per night-22 percent averaged at least seven hours and only 6 percent at least eight hours. About 71 percent had sleep quality ratings of greater than five, indicating poor sleep quality. The average HbA 1c level was 8.3 percent; 26 percent had a level below the recommended 7 percent.
Thirty-nine patients reported that their sleep was frequently disrupted by pain; these individuals were excluded from further analyses. Among the remaining 35 men and 87 women, 67 percent had poor sleep quality. Higher HbA 1c levels were associated with lower sleep quality, less sleep and a larger perceived sleep debt, even after researchers controlled for sex, age, BMI, complications and use of insulin. Participants were then classified based on whether or not they had complications from diabetes and whether or not they used insulin. "In patients without complications, perceived sleep debt but not subjective sleep quality was associated with lnHbA 1c levels," authors write. "In contrast, in patients with at least one complication, [sleep quality] score, but not perceived sleep debt, was a significant predictor after controlling for covariates."
The results do not indicate whether diabetes control impacts sleep-for instance, excessive urination at night resulting from high blood glucose levels could interrupt sleep-or whether insufficient or poor-quality sleep could contribute to poor glucose control. "Additional research is needed to determine whether optimizing sleep duration and quality may improve glucose control in patients with type 2 diabetes," conclude Dr. Knutson and colleagues. "Sleep curtailment has become increasingly prevalent in modern society, and it cannot be excluded that this behavior has contributed to the current epidemic of type 2 diabetes."
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