( Adolescents who are chronically exposed...)Family Turmoil and Violence Results in Stress-induced Physical Problems in Young, Cornell Psychologist Finds,Health

Adolescents who are chronically exposed to family turmoil, violence, noise, poor housing or other chronic risk factors show more stress-induced physiological// strain on their organs and tissues than other young people.

However, when they have responsive, supportive mothers, they do not experience these negative physiological changes, reports a new study from Cornell.

But the research group also found that the cardiovascular systems of youths who are exposed to chronic and multiple risk factors are compromised, regardless of their mothers' responsiveness.

The study, led by environmental and developmental psychologist Gary Evans, is published in the March issue of Developmental Psychology. It is the first study to look at how maternal responsiveness may protect against cumulative risk as well as the first, according to the researchers, to look at cardiovascular recovery from stress in children or youths.

Evans said that the findings suggest that the physiological toll of coping with multiple risk factors is significantly greater than with that of coping with a single event, even if that event was rather severe. "Moreover the burden appears to register in physiological systems that help us regulate our responses to stress," said Evans, the Elizabeth Lee Vincent Professor of Human Ecology and professor of human development and of design and environmental analysis in Cornell's College of Human Ecology.

To study stress-induced physiological changes in young teens, the researchers -- including three students who were undergraduates at the time and a graduate student -- used an index called allostatic load. This is a measure of stress-induced changes in neuroendocrine hormonal systems, cardiovascular responses and metabolism that indicate the severity of wear and tear that cumulative strain puts on organs and tissues.

"Allostatic load may very well turn out to be the primary mechanism of how risk, stre
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