www.Joyn7030.com, A New Web Site Dedicated to Helping Women Fight Joint Pain... ( Site Offers Advice for Joint-Pain Su...)

www.Joyn7030.com, A New Web Site Dedicated to Helping Women Fight Joint Pain, Launches

Date:10/17/2008

Site Offers Advice for Joint-Pain Sufferers Who are Confused and Concerned About Their Options for Fighting Joint Pain; Examines New Research That

Reveals Truth About Makeup of Joint Cartilage and Offers Hope

EGG HARBOR TOWNSHIP, N.J., Oct. 17 /PRNewswire/ -- A new Web site dedicated to helping women fight joint pain, http://www.Joyn7030.com, launched today. The site is designed to help joint-pain sufferers take control of their lives with a unique 70/30 joint-health plan. The site also examines new research that reveals the truth about the makeup of joint cartilage, provides expert opinions on why glucosamine has not met expectations in supporting joint health, and offers a place for women to connect and share ideas on living pain-free.

"Our goal is to help the many people suffering from joint pain live active, healthy, pain-free lives," said Leonard P. Smith, CEO, BSP Pharma, creator of the site. "Following the recent announcement from the National Center for Complementary and Alternative Medicine at the National Institutes of Health regarding the ineffectiveness of glucosamine and chondroitin in treating chronic joint conditions, many joint-pain sufferers are confused and concerned about their options. This site can help clear the confusion."

Because women have less joint cartilage than men, when it breaks down, it affects them in more places with more intensity. In fact, after menopause women experience a significant decline in joint collagen, which makes up 70 percent of cartilage and provides structure, strength and smoothness to the joint.

http://www.Joyn7030.com examines exciting new research that actually explains why glucosamine and chondroitin could not meet expectations as a comprehensive joint treatment because they are only 30 percent of the solution, and don't address the deterioration of Type II collagen,
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