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XRMD: Beyond AREDS2: Vision Improvement using Hormones, Genetics, Methylation, Nutrition and Anti-Oxidative Therapy
Date:5/16/2013

, a PhD in bio-organic chemistry and expert in unblocking methylation pathways concurs. "The impetus to only focus on anti-oxidation may have been a good idea in 2008 when the AREDS2 study began and we do employ anti-oxidative nutraceuticals, however in today's world of genetic bio-science there are more and better ways to optimize cellular health. We are finding that patients with macular degeneration have insufficient hormonal levels and problems in the methionine and transsulfuration pathway. This tells us that the retinal pigment epithelial cells, which become sick in macular degeneration, degenerate due to their inability to keep up with the tremendous demands placed on them within eye.

"In the early stage of macular degeneration, vision is still good and no improvement is possible", said Dr. Rozakis "Here the goal of therapy is stability. In late disease where the pigment epithelial cells and rods and cones are severely compromised or dead, no improvement is possible." The "marginal" macula with mild to moderate visual loss offers us an excellent opportunity to improve cellular function and measure a result."

"Do we have all the answers? Of course not, but the key is that we apply all the answers we have and not limit ourselves to anti-oxidative therapy as in AREDS2." said Dr. Rozakis. "Finding hormonal and genetic errors and being able to treat them is today's paradigm shift." As for rebuilding a severely degenerated macula, the hope is that stem cells can repopulate the pigment epithelium and that the rods and codes can reform as well. That is the future. In the meantime we must keep our existing pigment epithelial cells and rods and cones healthy using genetic knowledge, methylation science, hormones, anti-oxidation and nutrition all aimed at the cellular level." For more information contact Dr Rozakis at 440-777-2667 or visit xrmd.com.

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