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UC Davis researchers discover novel pathway to increased inflammation in diabetes patients
Date:11/27/2007

2 and TLR4 activity in type 1 diabetes monocytes, which are part of the bodys immune system, protecting against blood-borne pathogens by moving quickly to sites of infection. The immune system comprises the cells and mechanisms that defend the host from infection by other organisms, accomplishing its defense with the help of such pattern-recognition receptors as TLRs for early detection of specific classes of pathogens.

This finding provides us with a totally new insight into the causes of inflammation in diabetics, adds Jialal, who holds the Robert E. Stowell Endowed Chair in Experimental Pathology at UC Davis. Its an exciting development in the emerging area of TLR research that has potentially wide-ranging implications.

Further studies will use mice to examine the molecular mechanisms for increased TLR2 and TLR4 expression and determine their contribution to the pro-inflammatory state of diabetes.


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Contact: David Ong
david.ong@ucdmc.ucdavis.edu
916-734-9049
University of California - Davis - Health System
Source:Eurekalert

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