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Study identifies new risk factor for heart disease among kidney dialysis patients
Date:3/6/2013

in carbamylation. They also conducted experiments to test whether inducing amino acid deficiencies in animals led to increased protein carbamylation by urea. Finally, they conducted test tube experiments to investigate whether amino acids could directly inhibit protein carbamylation.

"Although dialysis therapy is life-saving and necessary to remove excess water and urea for patients with kidney failure, it also depletes patients' amino acids and other essential nutrients. In this way it removes both the good and the bad, and thus simply increasing patients' dialysis treatments will not necessarily reduce protein carbamylation," says Berg, adding that future studies will be needed to test whether amino-acid supplementation therapy reduces protein carbamylation and its associated risks, or if there are ways of modifying dialysis methods in order to remove urea without depleting amino acids.

"The mechanisms that lead to cardiovascular disease in patients with chronic kidney disease have remained elusive," says nephrologist Vikas Sukhatme, MD, PhD, Chief Academic Officer of BIDMC. "This paper provides an important first step in identifying kidney failure patients on dialysis at risk for cardiovascular disease and providing a rationale for treating them with inexpensive and non-toxic medicines such as amino acids."


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Contact: Bonnie Prescott
bprescot@bidmc.harvard.edu
617-667-7306
Beth Israel Deaconess Medical Center
Source:Eurekalert

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