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Scientist at The Fienstein Instute for Medical Research Discovers Quitting Smoking Promotes Anti-Inflammatory Response
Date:8/27/2009

ks, 30 percent of the women were not smoking. As they anticipated, several inflammatory markers, including tumor necrosis factor, soluble tumor necrosis factor receptors and soluble VCAM-1 (a marker of endothelial cell activation) dropped over time in 'quitters'. Dr. Metz and her colleagues found that those who were successful in quitting had lower C-reactive protein levels during the initial visit than those who continued to smoke despite the treatment. (Counseling was also provided)
   
"Maybe there are immunological differences in people who can quit and those who can't," said Dr. Metz. If she is right, measuring these inflammatory mediators early on and during the smoking cessation process may prove to be important in designing a treatment that be tailored to a person's biology. What's more, if smokers received their inflammatory mediator 'scores' during the quit process and saw some of these decline, it might be a strong incentive to stop smoking. She also stored genetic material from each smoker and will study the RNA and DNA that helps tell the story of the activity of immune markers.
   
The Staff at the Center for Tobacco Control and Dr. Metz also conducted another study funded by the New York State Department of Health on smokers with very early signs of chronic obstructive pulmonary disease. This new study included an additional smoking cessation drug, Chantix. They measured immune markers during the smoking cessation process and the results are being analyzed.

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Read the full story at http://www.prweb.com/releases/Feinstein_Medical/Quit_Smoking/prweb2780034.htm.


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