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Science Makes Inroads Against Prostate, Other Cancers
Date:4/19/2009

he explained. "It's almost a living organism. It's actually the patient's own T-cells -- white blood cells that exist in everyone's body -- that have been modified and engineered to be 'fooled' into attacking the cancer."

Junghans said that the first two patients to receive a single infusion with the new treatment experienced a 50 percent to 75 percent reduction in their blood level of prostate-specific antigen (PSA), a measure of prostate cancer activity, over the following two months.

"So, I'm very excited, because this achievement was with the lowest dose of the therapy, which we now plan to try out at a level ten times as high," he noted. Junghans said he and his team hope to "achieve a 100 percent reduction (in PSA)."

Another of the four studies included work led by Florence Hofman, a professor of pathology at the University of Southern California, Keck School of Medicine, in Los Angeles. Her team targeted brain cancer with an inhibitor called "dimethyl-celecoxib".

Celecoxib may sound familiar: It's the more scientific name for the cox-2 inhibitor drug, Celebrex, which has been used as a pain reliever and cancer-fighter but also has some cardiovascular side effects. The new drug is "similar to a cox-2, but not quite the same," Hofman noted. "It's like a key in a lock. You just change one of the little teeth in the key -- a molecule for example -- and it does one thing but not another, even if it looks very much the same. So then, we get the very great advantage of avoiding some known and dangerous side effects."

The therapy appears to inhibit new blood vessel growth as part of its strong anti-tumor effect, Hofman said. Specifically, in animal studies, this cox-2-like agent achieved a 35 percent to 45 percent reduction in the density of tumor blood vessels without the development of any potentially damaging long-term side effects.

Hofman and her colleagues noted that though their current focus was on
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