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Prostate cancer risk halved for subfertile men
Date:2/1/2012

ptor' because it mediates the harmful effects of the environmental toxicant dioxin, which can affect fertility.

If future research is able to more accurately determine which genes reduce the risk of prostate cancer, then this may open up new opportunities to develop drugs. However, before this can happen the genes with the desirable properties must be considered in a broader context, because reduced fertility is usually caused by a number of factors. The cancer-blocking properties must also be separated out and isolated from the properties that reduce fertility.

The researchers at Lund University are also interested in the reverse situation whether it is possible to find ways of helping men with reduced fertility by studying the genes of men with prostate cancer.


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Contact: Yvonne Lundberg
Yvonne.Giwercman@med.lu.se
46-705-977-904
Lund University
Source:Eurekalert

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