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New evidence caffeine may slow Alzheimer's disease and other dementias, restore cognitive function
Date:5/17/2010

ented memory deterioration and neurodegeneration in animal models of aging and of Alzheimer's disease."

Key findings presented in "Therapeutic Opportunities for Caffeine in Alzheimer's Disease and Other Neurodegenerative Diseases":

  • Multiple beneficial effects of caffeine to normalize brain function and prevent its degeneration

  • Caffeine's neuroprotective profile and its ability to reduce amyloid-beta production

  • Caffeine as a candidate disease-modifying agent for Alzheimer's disease

  • Positive impact of caffeine on cognition and memory performance

  • Identification of adenosine A2A receptors as the main target for neuroprotection afforded by caffeine consumption

  • Confirmation of data through valuable meta-analyses presented

  • Epidemiological studies corroborated by meta-analysis suggesting that caffeine may be protective against Parkinson's disease

  • Several methodological issues must be solved before advancing to decisive clinical trials

Mendona and Cunha also observe that "the daily follow-up of patients with AD has taught us that improvement of daily living may be a more significant indicator of amelioration than slight improvements in objective measures of memory performance. One of the most prevalent complications of AD is depression of mood, and the recent observations that caffeine might be a mood normalizer are of particular interest."


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Contact: Astrid Engelen
a.engelen@iospress.nl
31-206-883-355
IOS Press
Source:Eurekalert

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