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Mitochondria play role in pathogenesis of AD and estrogen-induced neuroprotection
Date:4/5/2008

s, or programmed cell death, of neurons of the brain;

  • and protection of neurons from mitochondrial toxins, which can induce further mitochondrial dysfunction and cell death.

    Dr. Brinton and her research team currently are validating the energy-production levels of mitochondria as a biomarker that could detect the presence of Alzheimers in the earliest stages, when the neurodegenerative process might be stopped or slowed or therapeutics be more effective. They believe this new information on how estrogen regulates mitochondrial function also sheds light on how to develop a new generation of effective Alzheimer therapeutics. Dr. Brinton currently is developing new, brain specific molecules that promote neurological defense against Alzheimers, using similar mechanisms as estrogen, but without estrogens negative side effects.


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  • Contact: Sylvia Wrobel
    ebpress@gmail.com
    770-722-1055
    Federation of American Societies for Experimental Biology
    Source:Eurekalert

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