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JCI table of contents: Sept. 4, 2007
Date:9/4/2007

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TITLE: Histone deacetylase degradation and MEF2 activation promote the formation of slow-twitch myofibers

AUTHOR CONTACT:

Eric N. Olson
University of Texas Southwestern Medical Center, Dallas, Texas, USA.
Phone: (214) 648-1187; Fax: (214) 648-1196; E-mail: eric.olson@utsouthwestern.edu.

View the PDF of this article at: https://www.the-jci.org/article.php?id=31960

RELATED MANUSCRIPT

TITLE: NO production results in suspension-induced muscle atrophy through dislocation of neuronal NOS

AUTHOR CONTACT:

Shin'ichi Takeda
National Center of Neurology and Psychiatry, Kodaira, Tokyo, Japan.
Phone: 81-42-346-1720; Fax: 81-42-346-1750; E-mail: takeda@ncnp.go.jp.

View the PDF of this article at: https://www.the-jci.org/article.php?id=30654

ACCOMPANYING COMMENTARY

TITLE: Two tales concerning skeletal muscle

AUTHOR CONTACT:

David J. Glass
Novartis Institutes for BioMedical Research, Cambridge, Massachusetts, USA.
Phone: (617) 871-7820; Fax: (617) 871-7049; E-mail: david.glass@novartis.com.

View the PDF of this article at: https://www.the-jci.org/article.php?id=33379


IMMUNOLOGY: Coming unstuck: T cell persistently tethered to integrin ligands can't reach their goal

For immune cells known as T cells to be effective they must home to the site of an infection. Similarly, for T cells to inappropriately mount an immune response and cause diseases such as colitis they must traffic to the site of the disease. The different integrin adhesion molecules expressed T cells regula
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Contact: Karen Honey
press_releases@the-jci.org
215-573-1850
Journal of Clinical Investigation
Source:Eurekalert

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