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JCI table of contents: April 25, 2011
Date:4/25/2011

ial Ca2+ determines the severity of inflammation in mouse lung microvessels

AUTHOR CONTACT:
Jahar Bhattacharya
Columbia University College of Physicians and Surgeons, New York, New York, USA.
Phone: 212.305.7093; Fax: 212.305.6724; E-mail: jb39@columbia.edu.

View this article at: http://www.jci.org/articles/view/43839?key=40f61efa7caa39f0b9d9

ACCOMPANYING COMMENTARY
TITLE: Mitochondrial Ca2+ and ROS take center stage to orchestrate TNF-alphamediated inflammatory responses

AUTHOR CONTACT:
Jacob I. Sznajder
Feinberg School of Medicine, Northwestern University, Chicago, Illinois, USA.
Phone: 312.908.7737; Fax: 312.908.4650; E-mail: j-sznajder@northwestern.edu.

View this article at: http://www.jci.org/articles/view/57748?key=e6d32124ce06b2bc0ee8


GASTROENTEROLOGY: Cellular connection between inflammatory bowel disease and colon cancers

Individuals with the inflammatory bowel disease ulcerative colitis are at high risk for developing colon cancer, but the mechanisms underlying this are not completely understood. A team of researchers, led by Stefan Fichtner-Feigl, at the University of Regensburg, Germany, has now identified a defined population of immune cells known as macrophages that facilitate the development of colon cancer in a mouse model of ulcerative colitis. These cells were marked by expression of F4/80, high levels of CD11b, and low levels of Gr1 and promoted tumor development, in part, via the production of the tumor-promoting factors IL-6 and TGF-beta-1. The authors plan to investigate whether individuals with ulcerative colitis also have increased numbers of these macrophages and whether their presence
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Contact: Karen Honey
press_releases@the-jci.org
734-546-5242
Journal of Clinical Investigation
Source:Eurekalert

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