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JCI online early table of contents: May 17, 2010
Date:5/10/2010

ller School of Medicine, Miami, has now provided new insight into the mechanisms regulating thyroid hormone action in the brain by modeling the process in an in vitro coculture system of human glioma cells and human neuroblastoma cells. Specifically, they find that human glial cells express a protein that increases the amount of active thyroid hormone available to act on nerve cells. Importantly, this pathway was activated in vivo in rat models of stroke and mouse models of inflammation. The authors therefore conclude that this pathway contributes to the regulation of thyroid hormone signaling in the brain during health and disease.

TITLE: Paracrine signaling by glial cellderived triiodothyronine activates neuronal gene expression in the rodent brain and human cells

AUTHOR CONTACT:
Antonio C. Bianco
University of Miami Miller School of Medicine, Miami, Florida, USA.
Phone: 305.243.5631; Fax: 305.243.7268; E-mail: abianco@med.miami.edu.

View this article at: http://www.jci.org/articles/view/41977?key=410293de40a91b71ef70


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Contact: Karen Honey
press_releases@the-jci.org
734-546-5242
Journal of Clinical Investigation
Source:Eurekalert

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