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JCI online early table of contents: July 12, 2010
Date:7/12/2010

which these signals are integrated during disease are unknown. Insight into this has now been provided by a team of researchers, led by Rama Khokha, at the Ontario Cancer Institute, Toronto, through their analysis of several strains of genetically engineered mice. Specifically, the team found that the proteins TIMP3 and ADAM17 cooperate to integrate the survival and death-inducing signals triggered by Fas activition during the acute liver stresses that can lead to fulminant hepatitis.

TITLE: Ectodomain shedding of EGFR ligands and TNFR1 dictates hepatocyte apoptosis during fulminant hepatitis in mice

AUTHOR CONTACT:
Rama Khokha
Ontario Cancer Institute, Toronto, Ontario, Canada.
Phone: 416.946.2051; Fax: 416.946.2984; E-mail: rkhokha@uhnres.utoronto.ca.

View this article at: http://www.jci.org/articles/view/42686?key=47ad1f09c331cff2def8


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Contact: Karen Honey
press_releases@the-jci.org
734-546-5242
Journal of Clinical Investigation
Source:Eurekalert

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