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JCI online early table of contents: July 1, 2011
Date:7/1/2011

g mechanism to explain the beneficial effect of Helicobacter pylori colonization on the development of allergen-induced asthma.

TITLE: Helicobacter pylori infection prevents allergic asthma in mouse models through the induction of regulatory T cells

AUTHOR CONTACT:
Anne Mller
University of Zrich, Zrich, Switzerland.
Phone: 41.44.635.3474; Fax: 41.44.635.3484; E-mail: mueller@imcr.uzh.ch.

Christian Taube
Johannes Gutenberg University, Mainz, Germany.
Phone: 49.6131.176848; Fax: 49.6131.176668; E-mail: taube@3-med.klinik.uni-mainz.de.

View this article at: http://www.jci.org/articles/view/45041?key=75a3d98b6d6b62150d82


ALLERGY AND ASTHMA: New axis of evil in asthma

Asthma is an extremely common inflammatory disease of the airways. Research performed in mice, by Stephen Galli, Mang Yu, and colleagues, at Stanford University School of Medicine, Stanford, has now identified a new aspect to the immune response underlying this condition. If the results hold true in humans, these data suggest a new therapeutic target.

An allergic asthma attack occurs when the immune system mistakenly identifies a harmless substance, such as cat dander, as a dangerous invader. The immune response associated with allergic asthma is classified as a Th2 response. However, recent data have suggested that this might be too simplistic a classification, as they have implicated the Th1 cellassociated soluble factor IFN-gamma as having a role. Galli, Yu, and colleagues confirmed that IFN-gamma has a role in a mouse model of chronic asthma that exhibits many features of severe asthma in humans. They also identified what it does: it triggers the activation of immune cells known as mast cells, which are critical for the develop
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Contact: Karen Honey
press_releases@the-jci.org
734-546-5242
Journal of Clinical Investigation
Source:Eurekalert

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