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JCI online early table of contents: Jan. 9, 2012
Date:1/9/2012

lleagues therefore suggest that targeting the CXCR4/CXCR7/SDF-1 pathway could provide a new approach to preventing and/or treating HUS associated with Escherichia coli O157:H7 infection.

TITLE: The CXCR4/CXCR7/SDF-1 pathway contributes to the pathogenesis of Shiga toxinassociated hemolytic uremic syndrome in humans and mice

AUTHOR CONTACT:
Philip A. Marsden
University of Toronto, Toronto, Ontario, Canada.
Phone: 416-847-1734; Fax: 416-864-5813; E-mail: p.marsden@utoronto.ca.

View this article at:
http://www.jci.org/articles/view/57313?key=94d0ea84d0b2f1f1dbba

LYMPHATIC SYSTEM: Role for RASA1 in regulating lymphatic vasculature growth and function

Capillary malformation-arteriovenous malformation syndrome (CM-AVM) is a disorder of the blood vessel network, which is known as the vasculature. Patients have a wide array of blood vessel abnormalities. Many of these are harmless but some can be life threatening. CM-AVM is caused by mutations in the RASA1 gene, which regulates blood vessel growth. However, it is not clear why some patients also develop symptoms that suggest that they have abnormalities in their network of vessels known as the lymphatic vasculature, which carries colorless fluid known as lymph around the body. A team of researchers led by Philip King, at the University of Michigan Medical School, Ann Arbor, has now provided a potential explanation for this they find that RASA1 regulates lymphatic vasculature growth and function in mice.

TITLE: RASA1 maintains the lymphatic vasculature in a quiescent functional state in mice

AUTHOR CONTACT:
Philip D. King
University of Michigan Medical School, Ann Arbor, Michigan, USA.
Phone: 734-615-9073; Fax: 734-764-3562; E-mail: kingp@umich.edu.


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Contact: Karen Honey
press_releases@the-jci.org
734-546-5242
Journal of Clinical Investigation
Source:Eurekalert

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