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JCI early table of contents for Nov. 8, 2013
Date:11/8/2013

on patterns may drive tumor formation. In this issue of the Journal of Clinical Investigation, MinGyu Lee and colleagues at MD Anderson Cancer Center evaluated histone methylation modifications in NSCLS cell lines and determined that the histone demethylase KDM2A was upregulated in NSCLC cell lines. Furthermore, induced KDM2A expression increased cell proliferation and invasiveness through activation of the ERK1/2 signaling pathway. Evaluation of tumors from NSCLS patients uncovered a correlation between increased levels of KDM2A and poor prognosis. This study suggests that therapies targeting KDM2A may benefit a subset of NSCLS patients.

TITLE: KDM2A promotes lung tumorigenesis by epigenetically enhancing ERK1/2 signaling

AUTHOR CONTACT: MinGyu Lee
MD Anderson Cancer Center, Houston, TX, USA
Phone: 713-792-3678; E-mail: mglee@mdanderson.org

View this article at: http://www.jci.org/articles/view/68642?key=c85b37ed02f837ee5ede


ALSO IN THIS ISSUE

TITLE: Antibodies against low-density lipoprotein receptorrelated protein 4 induce myasthenia gravis

AUTHOR CONTACT: Lin Mei
Institute of Molecular Medicine and Genetics, Augusta, , USA
Phone: 7065138002; E-mail: lmei@georgiahealth.edu

View this article at: http://www.jci.org/articles/view/66039?key=c1820c1751a31705f85c

TITLE: Inhibition of Coxsackievirus-associated dystrophin cleavage prevents cardiomyopathy

AUTHOR CONTACT: Kirk Knowlton
UCSD, La Jolla, CA, USA
Phone: 858-822-1364; Fax: 858-822-3027; E-mail: kknowlton@
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Contact: Corinne Williams
press_releases@the-jci.org
Journal of Clinical Investigation
Source:Eurekalert

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