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JCI early table of contents for Nov. 26, 2012
Date:11/26/2012

ice, Orser and colleagues found that the drugs inhibited the activity of glycine receptors in the brain, leading to seizures. Seizures could be prevented by co-treatment with the general anesthetic isoflurane. This study explains the causes of and proposes treatment for antifibrinolytic-induced seizures. In a companion commentary, Debra Schwinn of the University of Washington reviews the connection between seizures and antifibrinolytic drugs.

TITLE:
Tranexamic acid concentrations associated with human seizures inhibit glycine receptors

AUTHOR CONTACT: Irene Lecker
Faculty of Medicine, University of Toronto, Toronto, ON, CAN
Phone: 416-978-1518; E-mail: irene.lecker@utoronto.ca

View this article at: http://www.jci.org/articles/view/63375?key=806b0eca4232a067ca57

ACCOMPANYING COMMENTARY

TITLE:

Understanding the TXA seizure connection

AUTHOR CONTACT:
Debra Schwinn
University of Washington, Seattle, WA, USA
Phone: 206-543-2673; Fax: 206-543-2958; E-mail: debra-schwinn@uiowa.edu

View this article at: http://www.jci.org/articles/view/66724?key=bd9895d06f784ef4366f


Parallel structure: Surprising similarities between kidney cells and neurons

The primary function of the kidneys is to filter the blood to remove waste and retain blood cells and proteins. Podocytes are specialized kidney cells that form a filtering structure known as a slit diaphragm. Disruption of the podocytes results in the enlargement of the slit diaphragm, causing nephrotic syndrome, and, eventually, renal failure. In this issue of the Journal of Clinical Investigation, researchers led by Shuta
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Contact: Jillian Hurst
press_releases@the-jci.org
Journal of Clinical Investigation
Source:Eurekalert

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