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JCI early table of contents for Nov. 19, 2012
Date:11/19/2012

rchers at New York University to hypothesize that factors elicited by HIV-1 might block the function of DCs. In this issue of the Journal of Clinical Investigation, Bhardwaj and colleagues report that microparticles found in the plasma of HIV-1 patients inhibited the function of normal DCs and induced dendritic cell death. These results suggest that inhibition of these microparticles could promote DC activity and survival and help fight HIV-1.

TITLE:
HIV-1 infectioninduced apoptotic microparticles inhibit human DCs via CD44

AUTHOR CONTACT:
Nina Bhardwaj
NYU School of Medicine, New York, NY, USA
Phone: 1 212 263 5814; Fax: 2122636729; E-mail: nina.bhardwaj@nyumc.org

View this article at: http://www.jci.org/articles/view/64439?key=1d6cff10286e34fcfebf


B cells cross the blood-brain barrier in multiple sclerosis

In multiple sclerosis (MS), the blood-brain barrier (BBB) is compromised, allowing immune cells to attack the central nervous system (CNS). B cells are a population of immune cells that are thought to trigger autoimmune responses in MS, but it is unclear if they are able to transverse the BBB. In this issue of the Journal of Clinical Investigation, researchers led by Hans-Christian von Bdingen at the University of California, San Francisco, studied the ability of B cells to move between the peripheral blood and the CNS. By comparing the antibodies in the cerebrospinal fluid and the blood stream, von Bdingen and colleagues found populations of B cells that are common to both areas in patients with MS. This study demonstrates that B cells can cross the BBB and shows that they are central regulators of the pathological immune response in MS.

TITLE:
B cell exchange across the blood-brain barrier in multiple sclerosis

AUTHOR CONTACT:
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Contact: Jillian Hurst
press_releases@the-jci.org
Journal of Clinical Investigation
Source:Eurekalert

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