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Insomnia after myocardial infarction
Date:12/1/2010

ompanied by insomnia, we wanted to verify whether the neurons in the brainstem were also affected," the investigator explained.

As demonstrated in this study, myocardial infarction, in addition to causing depression, is also associated with the release of factors that provoke the inflammation of tissues, including the brain, and specifically the regions that control sleep, notably the paradoxical sleep phase. The particular function of that phase is to activate regions in the brain that are responsible for integrating our emotions. If that is affected, the risk of depression also increases.

Poor-quality sleep is a known risk factor for cardiovascular disease. Since it can affect remission after an infarction, the risk of complications and recidivism rises and a vicious circle may be set in motion.

Godbout says this study illustrates the importance of rapid intervention in the days following the infarction, before the first signs of insomnia and depression are even apparent. He notes that "any preventive, pharmacological or behavioural treatment is certainly a pathway that should be considered."


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Contact: Vronique Allaire
veronique.allaire.hsc@ssss.gouv.qc.ca
514-338-2222 x2473
University of Montreal
Source:Eurekalert

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