Cancer Mortality Increases With Cancer Cell Repl...(s than expected from their Replikin Coun...),Health

Date:12/4/2008

s than expected from their Replikin Counts (ie shifted to the left), possibly because of the improvement in their early detection and prompt treatment.
 
To obtain these results, all cancer protein sequences published on PubMed were analyzed by histological cancer cell type by ReplikinForecast™ software. The Replikin Peak Gene (RPGene)* was identified in each cell type. The replikins in each RPGene were counted (Replikin Count = number of replikins per 100 amino acids).  The Replikin count of each RPGene then was compared in the different histological cancer cell types as shown in the attached Figure.
 
Replikin count has already been shown by Replikins, Ltd. to be related to rapid replication, disease outbreaks, and host mortality in viruses such as influenza and HIV, bacteria, malaria, and other infectious diseases*. The relationship between Replikin count and mortality in cancer parallels that found in H5N1 virus in humans and in Taura Syndrome Virus in shrimp.
 
The authors speculate that in low mortality cancers, it is possible that the immune system may be adequate to the number of invading units; but that the increased mortality rate in viruses and in cancer may reflect the overwhelming of host immune defenses by markedly increased numbers of viruses or of cancer cells produced in each case by increasing replikin concentration in the up-regulated Replikin Peak Gene.  An increase in number of cancer cells per unit time produced by rapid replication has been demonstrated in the laboratory in the case of glioblastoma multiforme brain cancer cells in tissue culture*. The quantitative relationship of Replikin Count to mortality rate has been demonstrated in the laboratory in four strains of lethal Taura Syndrome Virus in shrimp*.
 
These findings have diagnostic, therapeutic, and preventive applications. On the diagnostic side, 1) Oncolab, Inc.'s AMAS®Test, FDA permitted to market and Medicare
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