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Cancer Mortality Increases With Cancer Cell Replikin Count
Date:12/4/2008

Drs. Samuel and Elenore Bogoch of Oncolab, Inc. have found that the concentration of replikins, a new class of peptides in genomic proteins*, are quantitatively related to the mortality rate in human cancer of different cell types. No cancer cell genomic structure has previously been reported to be quantitatively related to mortality rate.

Boston, MA (PRWEB) December 4, 2008 -- Drs. Samuel and Elenore Bogoch of Oncolab, Inc. have found that the concentration of replikins, a new class of peptides in genomic proteins*, are quantitatively related to the mortality rate in human cancer of different cell types. No cancer cell genomic structure has previously been reported to be quantitatively related to mortality rate.


     
The attached figure shows the relationship between the Replikin Count™ in the Replikin Peak Gene* and the percent mortality in human cancer: the higher the Replikin Count, the higher the percent mortality after 5 years.  Glioblastoma multiforme and non-small-cell lung cancer, the two cancer cell types with the greatest 5-year mortality rates in 2002 (98% and 91% respectively), are also the cell types which have the highest Replikin Counts (325 and 250 respectively).
 
The count in Glioblastoma Multiforme is 18 times higher than the counts in prostate and thyroid cancer which have the lowest Replikin Counts (20 and 15 respectively) as well as the lowest 5-year mortality rates (each approximately 2%). The quantitative relationship of Replikin count to mortality is also seen in the figure for other common cancers, which shows some deviations from an 'ideal' curve. For example, gastric cancer has a higher mortality rate than expected from its Replikin Count (ie shifted to the right), possibly because of the known difficulty of early detection of gastric cancer. Breast and urinary bladder cancer have lower mortality rate
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