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Alzheimer's Plaques More Complex Than Thought
Date:2/26/2009

ctivity wasn't reduced when the researchers blocked the activity of neurons. This indicates that the known impact of amyloid plaques on neurons isn't responsible for increased astrocyte activity.

"This is the first clear evidence in a live animal model that amyloid plaques perturb calcium signaling across the astrocyte network via a neuron-independent mechanism," Kuchibhotla said. "It has been suggested that these intercellular calcium waves, which previously had been observed only in response to some sort of external stimulus, indicate the existence of, or response to, a traumatic insult. Our data support this hypothesis, but whether the calcium signals we observed actually protect or harm cells remains to be determined."

"One key question will be how increased astrocyte signaling impacts neuronal function, and another will be whether astrocyte activity limits or intensifies plaque deposition," Kuchibhotla added.

More information

The U.S. National Institute on Aging has more about Alzheimer's disease.



-- Robert Preidt



SOURCE: Massachusetts General Hospital, news release, Feb. 26, 2009


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Alzheimer's Plaques More Complex Than Thought
Alzheimer's Plaques More Complex Than Thought
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