Lactic acidosis

Lactic acidosis
Definition:
Lactic acidosis occurs when lactic acid accumulates in the blood stream at a faster rate than it can be eliminated. Lactic acid is produced when oxygen levels in the body drop. The most common cause is intensive exercise.
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Lactic acidosis: Acidosis (too much acid in the body) due to the buildup of lactic acid in the body. Lactic acidois occurs when cells make lactic acid (from glucose) faster than it can be metabolized.
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LACTIC ACIDOSIS: High levels of lactic acid in the blood, which is potentially fatal. Symptoms include nausea, vomiting, fatigue, and shortness of breath. The use of nucleoside analogs can contribute to lactic acidosis.
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Lactic acidosis can be caused by taking large doses of acetaminophen and alcohol and by intravenous infusion of epinephrine, glucagon, fructose, or sorbitol. Antifreeze poisoning can also cause lactic acidosis.
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Effect of coenzyme Q10 in patients with mitochondrial myopathy, encephalopathy, lactic acidosis, and stroke-like episodes (MELAS): evaluation by noninvasive tissue oximetry. J Neurol.Sci. 1-1-1999;162(1):65-68.
Akihama, T., Nakamoto, Y., Shindo, T.
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Lactic acidosis
Lactose intolerance
Laennec's cirrhosis
Lambert-Eaton Syndrome
Lamellar ichthyosis
Lancefield group B streptococcus
Landouzy-Dejerine muscular dystrophy
Landry-Guillain-Barre syndrome
Langerhans cell histiocytosis ...
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As the disorder progresses, symptoms may also include generalised weakness, lack of muscle tone, and episodes of lactic acidosis. Lactic acidosis, the accumulation of lactic acid in the brain, may lead to impairment of breathing and kidney function.
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Lactic acidosis is an accumulation of lactic acid. This can be caused by many conditions, including prolonged lack of oxygen, certain diseases, prolonged exercise, hyperventilation, and medications such as oral medications used to treat diabetes.
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Labyrinthitis
Lack of Intrinsic Factor
Lacrimal Gland Tumor
Lactase Deficiency (Lactose Intolerance)
Lactic Acidosis ...
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It is currently in phase III clinical trials in patients with closed head injury and in phase II trials related to the treatment of stroke patients and the treatment of pediatric patients suffering from congenital lactic acidosis.
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