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NFCR Scientists Discover Brain Tumor's “Escape Path”
Date:1/28/2010

NFCR Scientists Webster Cavenee, Ph.D., and Frank Furnari, Ph.D., along with their team at the Ludwig Institute for Cancer Research at the University of California San Diego School of Medicine, have recently discovered one way that brain tumors manage to escape an attack launched by certain “smart drugs” designed to stop tumor growth. This breakthrough discovery revealed a new cellular mechanism that could be used to develop a more effective treatment strategy for malignant gliomas, the most common type of primary brain tumors.

Bethesda, MD (PRWEB) January 28, 2010 -- National Foundation for Cancer Research Scientists Webster Cavenee, Ph.D., and Frank Furnari, Ph.D., along with their team at the Ludwig Institute for Cancer Research at the University of California San Diego School of Medicine, have recently discovered one way that brain tumors manage to escape an attack launched by certain “smart drugs” designed to stop tumor growth. This breakthrough discovery revealed a new cellular mechanism that could be used to develop a more effective treatment strategy for malignant gliomas, the most common type of primary brain tumors.

Scientists have long searched for a new way to treat malignant gliomas, which are difficult to treat with traditional therapies, such as surgery, radiation and chemotherapy. In recent years, new treatments have been developed to improve a patient’s treatment outcome, but these treatments are less effective when used to treat malignant gliomas. These new treatments, called targeted therapies, target tumor-specific molecules in the body. Yet, when used on malignant gliomas, the results are disappointing. For instance, some “smart drugs” are designed to hit a tumor target called Epidermal Growth Factor Receptor (EGFR). However,
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