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JCI table of contents: Dec. 3, 2007

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TITLE: Mutations in the EGFR kinase domain mediate STAT3 activation via IL-6 production in human lung adenocarcinomas

Jacqueline F. Bromberg
Memorial Sloan-Kettering Cancer Center, New York, New York, USA.
Phone: (212) 639-2577; Fax: (646) 422-2231; E-mail:

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TITLE: IL-6 involvement in epithelial cancers

Joan S. Brugge
Harvard Medical School, Boston, Massachusetts, USA.
Phone: (617) 432-3974; Fax: (617) 432-3969; E-mail:

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HEMATOLOGY: G1ME more platelets: An inflammatory mediator drives maturation of platelet precursor cells

Infection and inflammation are sometimes accompanied by an excess of circulating platelet cells, a condition called thrombocytosis. In a new study, John Crispino and his colleagues at Northwestern University Feinberg School of Medicine, Chicago, have explored this relationship between inflammatory disease and platelet formation in mice.

Cellular differentiation is the process by which cells specialize. For example, megakaryocytic precursor cells differentiate into megakaryocytes, the parent cells of platelets. G1ME cells are mouse megakaryocytic precursors tha

Contact: Karen Honey
Journal of Clinical Investigation

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