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Gladstone scientists identify role of tiny RNAs in controlling stem cell fate
Date:3/5/2008

and other organisms. Activity of either miR-1 or miR-133 in ES cells caused genes that encourage mesoderm formation to be turned on. Equally important, they caused other genes that would have told the cell to become ectoderm or endoderm to turn off. For example, expression of a specific factor called Delta-like 1 was repressed by miR-1. Removal of this factor from cells by other methods also caused the cells to begin transforming into heart cells.

Our findings provide insight into the fine regulation of cells and genes that is needed for a heart to form, said Kathy Ivey, PhD, a California Institute of Regenerative Medicine (CIRM) postdoctoral fellow and lead author on the study. By better understanding this complicated system, in the future, we may be able to identify ways to treat or prevent childhood and adult diseases that affect the heart.


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Contact: Valerie Tucker
vtucker@gladstone.ucsf.edu
415-734-2019
Gladstone Institutes
Source:Eurekalert

Page: 1 2

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