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23ème congrès européen sur l'Hypertension et la Protection Cardiovasculaire
Date:6/25/2013

lope, Chef de l'Unité Hypertension à l'Hôpital du 12 Octobre de Madrid en Espagne, et investigateur principal de l'étude SEVITENSION. "Les études post-commercialisation telles que celle-ci fournissent des données importantes pour aider les médecins à choisir les meilleures options thérapeutiques afin de prendre en charge de manière plus efficace et plus pratique l'hypertension artérielle, et ainsi de mieux protéger les patients. Cette amélioration potentielle dans la prise en charge de l'HTA aidera les médecins à atteindre l'objectif ambitieux de contrôler la pression artérielle chez au moins 70% des patients hypertendus d'ici 2015."

Les médicaments tels que les inhibiteurs de l'enzyme de conversion de l'angiotensine (IEC) réduisent la pression artérielle en inhibant l'activité du système rénine-angiotensine (SRA). Une classe plus récente de médicaments antihypertenseurs, appelés antagonistes du récepteur de l'angiotensine (ARA II) réduit aussi la pression artérielle en inhibant le SRA, et offre l'avantage de provoquer moins d'effets secondaires que les IEC, notamment la toux.[7]

Daiichi Sankyo soutient activement la recherche sur les antihypertenseurs, et a développé toute une gamme de médicaments à base de l'ARA II olmésartan. Un de ces produits, l'association fixe olmésartan/amlodipine (Sevikar®), a été utilisée pour tenter de répondre à une question clinique importante : est-ce que l'association de deux médicaments composée d'un ARA II tel que l'olmésartan, et de l'amlodipine peut réduire la pression artérielle centrale aussi efficacement que l'association du périndopril et de l'amlodipine ? Cette question a &#
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SOURCE Daiichi Sankyo Europe GmbH
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