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U-M scientists find genes that control growth of common skin cancer

oncogene is frequently followed by the emergence of malignancies that have activated alternative cancer-causing pathways. According to Hutchin, the fact that this does not happen in regressed mouse BCCs suggests that any residual human BCC cells, if present, should also remain in check as long as the Hh pathway is effectively inhibited. Funding for this research was provided by grants from the National Cancer Institute and the National Institute of Arthritis and Musculoskeletal and Skin Diseases.
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Source:U-M Medical School


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