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Turning a cellular sentinel into a cancer killer

e therapeutic benefits."

Lowe emphasized that these new experimental techniques for reactivating p53 could also be applied to study the effects of other regulatory molecules on cancers. "We think we have a really powerful technology that could be generalized, not only to study tumor suppressors, but also molecules that might be important drivers of tumorigenesis, making them good drug targets," he said. "For these, we could do the converse experiment, letting the tumor develop and then using our technique to knock out the molecule and see whether the tumor goes away."

Jacks and his colleagues next plan to study the effects of p53 reactivation in other cancers. They hope to use the technology to understand why some cancers respond by undergoing apoptosis, while others undergo senescence.


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Source:Howard Hughes Medical Institute


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