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To Stop Evolution: New Way Of Fighting Antibiotic Resistance Demonstrated By Scripps Scientists

experiments, the rodents were given rifampicin or ciprofloxin, and the scientists found that nearly all the wild-type E. coli cells had grown fully resistant to the antibiotics after three days.

This research is also significant for cancer because cancer cells mutate readily and often acquire resistance to common chemotherapies. Romesberg hopes that if he and his colleagues can identify the molecules that human cancer cells use to drive evolution, they can find ways of intervening and preventing the evolution of chemotherapy resistance in these cancer cells. Last year Romesberg was awarded a Department of Defense Breast Cancer Research Program Idea Award of $563,100 to identify the genes in Saccharomyces cerevisiae yeast and human cells that induce mutations that can transform normal cells into cancer cells.

The article, "Inhibition of Mutation and Combating the Evolution of Antibiotic Resistance" by Ryan T. Cirz, Jodie K. Chin, David R. Andes, Valöée de Crï'-Lagard, William A. Craig, and Floyd E. Romesberg appears in the June, 2005 issue of the journal PloS Biology. See: http:/?​dx.doi.org/?0.1371/​journal.pbio.0030176


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Source:Scripps Research Institute


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... to LYZL6 ( Abpromise for ... Antigen: Synthetic peptide: DWLCL AFVE ... corresponding to amino acids 46-100 of ... Gene ID: 57151 ...
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Mouse monoclonal [DJR3] to DcR1 (Phycoerythrin) ( Abpromise for all tested applications). entrezGeneID: 8794 SwissProtID: O14798...
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