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Scientists reverse evolution

d on a possible example: a form of inherited anemia named beta-thalassemia, which occurs when a mutant beta hemoglobin gene results in faulty production of hemoglobin. The new study suggests it might be possible to use regulatory sequences from the faulty gene to activate a similar gene, essentially creating an embryonic or juvenile beta hemoglobin gene that could make hemoglobin normally during adult life.


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Source:University of Utah Health Sciences Center


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