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Scientists observe how a close bond activates the immune system

est when they are physically held in the outer ring of the bull's eye rather than the center.

"We locked the receptors in the periphery and saw enhanced signaling over a prolonged period of time. It was quite a surprise," says Dr. Dustin. Researchers had speculated that the concentrated bull's eye structure somehow allowed T cells to maintain their state of activation. But the new work shows that it is actually the outer edge of immunological synapse that boosts activation, not the center.

Dr. Dustin's group is now conducting additional experiments to see if dendritic cells actively present proteins to T cells in patterns that stimulate the periphery of the bull's eye in the immunological synapse, using molecular organization to provide information about the precise nature of the threat associated with the antigen.


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Source:New York University Medical Center and School of Medicine


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