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New compound reduces stroke damage

strongly inhibits FKBP38 leaving other brain FKBP almost untouched under certain conditions," said Fischer. "A strong neuroprotective effect became obvious in an animal model of stroke when the animals were treated with this lead compound."

Fischer and his colleagues found that their compound protected the rats' neurons and also caused neural stem cell proliferation and neuronal differentiation. Diseased animals with motor behavior deficits also showed improvement when they were given the synthetic drug.

These results suggest a potential therapeutic application specific FKBP38 inhibitors in the treatment of neurodegeneration following stroke and a number of other diseases.


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Source:American Society for Biochemistry and Molecular Biology


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